sciencehabit shares a report from Science Magazine: For more than half a century, physicists have searched for one of the strangest particles predicted by modern theory. It would be made almost entirely of gluons, the elusive subatomic particles that carry the strong nuclear force. Now, using a particle collider in Beijing, researchers say they have effectively proved the existence of such a "glueball." In a preprint posted last month on arXiv and in a presentation last week at the International Conference on High Energy Physics (ICHEP), the researchers argue that a particle called X(2370), which they produced by smashing electrons into positrons at high energies, has the properties expected of a glueball. "It's an experimental triumph," says Colin Morningstar, a particle physicist at Carnegie Mellon University who was not involved in the finding. "It's the strongest evidence yet that particles dominated by a glueball component can exist in nature."

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Researchers in China have developed a living mycelium textile that can self-clean, renew its surface, and partially repair holes when treated with a nutrient solution and fresh fungus. The material can also gain added properties such as blue pigmentation or UV resistance by co-culturing it with yeast or other fungi. Dezeen reports: The breakthrough from the researchers at the Shenzhen Institutes of Advanced Technology is a type of engineered living material (ELM) -- a material built off living organisms that stay active even after they're fabricated into their final form. [...] By working with living but dormant cordyceps militaris fungus instead, the researchers have been able to take advantage of its biological functions. The result is a material that is self-renewing and responsive to its environment, in ways that could one day transform architecture and clothing -- as seen in a prototype dress created together with material innovation company Peelshere. It can also be adapted by mixing in other fungi or yeast, lead researcher Ke Li and her team detail in a paper in the peer-reviewed journal Science Advances. In it, they describe a "programmable fungal platform" where mycelium is treated like a modular system, with the sheet material forming a base structure and extra biological abilities, such as colour and UV resistance, becoming "plug-and-play" add-ons via other organisms. This gets their textile closer to the self-repair, environmental responsiveness and controllable functionality that is the promise of engineered living materials, they argue. The ELM's self-renewing and semi-repairing functionality comes from the mycelium base structure. Following drying at 45 degrees, the material is not quite living and not quite dead, but instead in a "low-metabolic, dormant-like state", Li told Dezeen, meaning it is not actively growing. However, new growth can be triggered by applying a nutrient solution of potato water, leading the dormant mycelium to germinate, send out new fungal filaments and renew the material's surface. When this nutrient solution is applied over a hole, along with a small patch of fresh fungus, it triggers the living cells to grow across the gap, seamlessly repairing the surface without any adhesives or stitching. The material is also naturally self-cleaning, as it is hydrophobic. "Its distinctive surface texture, biological colouring, controlled repair and biodegradability may be particularly useful in applications where visual expression and a defined product lifetime are important," said Li. "Further improvements in durability, moisture resistance, safety and manufacturing consistency would be needed before it could be considered for routine clothing or permanent architectural use."

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"A major scientific review is challenging the idea that more protein is always better," reports ABC News: The review, led by pathologist and biomedical researcher Dudley Lamming and published in the journal Cell Press Blue, examined more than 350 studies involving humans, mice, insects, yeast, and other organisms. The review found that eating less protein, or less of certain amino acids that make up protein, may turn on body processes linked to healthier aging, as well as metabolism, the process by which the body turns food and drinks into energy... For adults who already get enough, eating more protein may offer little benefit, while some research suggests that eating less protein could support healthier aging... Lamming reviewed the effect of reducing protein in many body processes. One of the processes involves a hormone called FGF21. "There's an increase in a hormone called FGF21 that promotes energy expenditure [when protein intake is reduced]," Lamming said. "It essentially increases thermogenesis in your adipose tissue, so you don't just store fat, but actually burn it as fuel." This may help explain why some studies connect lower-protein diets with less body fat, better blood sugar control and a healthier metabolism. Eating less protein may also affect signals that tell cells when to grow, repair damage or recycle old cell parts. These jobs may play a role in aging. However, protein restriction is not the same as protein deficiency. The goal is not to deprive the body of an essential nutrient. Instead, the research raises the possibility that avoiding unnecessary excess could benefit some people who already consume enough... Before reaching for another high-protein product, a better question may be: How much protein does my body actually need?

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alternative_right shares a report from ScienceAlert: A study published in PLOS One in 2021 by researchers from Durham University in the UK showed that with 10 weeks of training, both blind and sighted people could learn to echolocate using verbal clicks. While the technique is already used by a number of people with impaired vision -- sometimes using the taps of a cane instead of clicks made by their mouth -- those findings suggest that many of us can learn the necessary techniques. Some of the same researchers who made that discovery are part of the team behind a follow-up study published in Cerebral Cortex, looking at how the 10 weeks of training that the 26 participants went through actually changed the physical structure of their brains. Specifically, the team analyzed brain scans of the V1 (the primary visual cortex, processing visuals), and the A1 (the primary auditory cortex, processing sounds). Strikingly, the scans showed that the V1s of both blind and sighted people had developed sensitivity to sound echoes during echolocation training.

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An anonymous reader quotes a report from CBC News: It's rare that cancerous tumors can spread from one individual to another. But a genetic study suggests that's what's happening with melanoma tumors among catfish in Quebec and the northeastern U.S. The discovery represents the first known transmissible cancer in fish and one of very few transmissible cancers ever found, reported the study published in the journal Nature on Wednesday. Julie Dragon, co-leader of the new study, noted that only three other transmissible cancers have ever been identified so far -- in Tasmanian devils, dogs and shellfish. "These conditions are incredibly rare in nature," she said. "So something has to be happening to allow this to happen." Anglers in Lake Memphremagog, which spans the border between Quebec and Vermont, first reported catching brown bullhead catfish with strange black spots and lumps in 2012. They turned out to be melanoma skin cancer tumors. (Humans can also get this kind of cancer.) Now, about a third of the brown bullheads living in the lake have them. It's not clear how sick the fish become. In some cases, the cancer spreads to other organs, such as the brain or liver. But many fish with lesions seem relatively healthy and some older fish even have them, suggesting they can live with the disease for a time. Because of the sudden appearance of the black lesions in Lake Memphremagog the year after a huge flood caused by post-tropical storm Irene, locals worried they were caused by carcinogens washed into the lake by floodwaters. Tests for carcinogens haven't been conclusive, although a study published in May found higher concentrations of seven metals, including zinc and the carcinogen arsenic, in the skin of fish with the tumors. Researchers suspect the tumors may spread among adult fish during spawning, when crowded fish rub against one another and may be punctured by their spines. "They... swim all over each other and we think it's possible that they poke each other and cells can get into other fish that way," Dragon said, though transmission has not yet been demonstrated.

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A new study by researchers at the University of California at San Francisco "adds to growing research linking increased social media use to detrimental effects on attention, memory and cognition," reports the Washington Post: The study followed more than 11,000 U.S. adolescents over a period of five years, with participants first asked about their own social media use at the average age of 12, and surveyed annually through the average age of 16. Researchers found that increases in addictive social media use were followed by rising ADHD one year later — particularly among boys who reported rising addictive social media use at ages 14 and 15. This association was not found consistently in reverse, meaning that ADHD symptoms did not appear to precede higher levels of addictive social media use... "When an individual adolescent's addictive social media use score increased from one year to the next, that same adolescent tended to show an increase in ADHD symptoms in the following year...." [said Jason Nagata, lead author of the study and an associate professor of pediatrics at the University of California at San Francisco]. He urged parents to consider: "Can their kids stop if they want to? Is social media interfering with their schoolwork? Is it impairing their social relationships? Are there addiction-like symptoms, like withdrawal and relapse?" Approximately 7 million American children between the ages of 3 and 17 have received an ADHD diagnosis, according to the Centers for Disease Control and Prevention, and boys are diagnosed with ADHD at about twice the rate of girls. The study did not find a clear link between addictive social media use and ADHD among girls, Nagata said. "Some studies do suggest that teenage boys in particular may be more sensitive to immediate reward and sensation-seeking in adolescence," he said. And social media platforms are designed to provide exactly that: "It encourages frequent task-switching, and there's this constant stream of stimulation that might make it harder for adolescents to maintain and sustain attention that is needed for schoolwork and daily life," he said. "The design features of social media offer the constant reinforcement of impulsivity — it offers immediate gratification and novelty and it encourages multitasking, which can then override working memory and executive control." Experts have long noted that this kind of digital exposure is particularly significant during critical stages of mental, social-emotional and cognitive development... [I]t's especially important for parents themselves to demonstrate a healthier relationship with screens and social media. "One of our previous findings was that parental screen use is a very strong predictor of kids' screen use," Nagata said.

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The Verge argues that researchers "have made genuine progress in quantum computing — it's just been largely incremental and too esoteric to immediately capture the public's imagination." And there are predictions that quantum computers will finally do something useful as soon as 2028: The drama can overshadow the real progress in quantum computing... Researchers have improved the qubits themselves, so they hold onto information longer. When they hold onto information longer, you can fit in more operations and do more complicated algorithms. Last November, Andrew Houck of Princeton University and his colleagues reported that they'd made a superconducting qubit that can hold onto information three times longer than the previous record holder... And in the last two years, researchers have made substantial strides in what's known as quantum error correction... In addition, researchers have developed algorithms to correct errors while the quantum computer operates... Microsoft claimed, which experts dispute, that it made an object made of electrons known as a Majorana particle [which should make fewer errors and be easier to scale up]... "We 100 percent stand behind our results. We stand by our roadmap," Microsoft's quantum lead, Chetan Nayak, responded in an interview with The Verge. In an email statement, he added that Microsoft's "papers do show that we are creating and controlling Majorana [particles]... Microsoft's supporting evidence is unconvincing [according to [Henry Legg, a physicist from the University of St. Andrews and a longtime Microsoft critic]Rnqyq. What it claimed as evidence of a Majorana particle, he says, could actually be due to quantum dots forming in its device. Quantum dots are electron-containing objects that are not useful for Microsoft's quantum computer. It also bases its claim on data from a single device, says Legg. He wants to see Microsoft replicate the results in multiple chips. "If you repeatedly try and find Jesus in your toast, eventually you'll find Jesus in your toast," he says. "But that one piece of toast doesn't mean you had some kind of epiphany." "While we appreciate the religious fervor, our data maintains the strength and consistency of our roadmap, as we have for the past several years across previous milestones. We look forward to delivering the world's first quantum machine and sharing the energy of our achievements with the world," wrote Nayak in response. Past spurious work from Microsoft-affiliated researchers adds to the doubt. In 2021, the journal Nature retracted an article from Microsoft-affiliated researchers in which they'd claimed strong experimental evidence that they'd created a Majorana particle. "Even hopeful experts have varying opinions about when a quantum computer will demonstrate something useful," the article acknowledges. But quantum computing lecturer Eleanor Crane of King's College London predicts researchers will have demonstrated a useful scientific simulation on a quantum computer by 2028. Thanks to Slashdot reader joshuark for sharing the article.

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Anthropic has launched Claude Science, an AI workbench that connects more than 60 scientific databases and tools through a single interface. Through the platform, Basecamp Research is making its EDEN models available for tasks such as designing antibiotic peptides and predicting vaccine targets from simple text prompts, though the results still require laboratory testing before clinical use. Genetic Engineering and Biotechnology News reports: In a Claude Science demo, Oliver Vince, PhD, co-founder at Basecamp, uploaded a sample patient microbiology report. When given a simple natural language prompt, the platform designed peptides, predicted their efficacy, and provided a shortlist of candidates most likely to succeed in experiments in minutes. While generating human-ready antibiotics at the click of a button is still a step away, Vince said democratizing these tools is a powerful first step, particularly for researchers in regions where accelerated computing infrastructure is not readily accessible. "Most models require you to be a computational scientist," Vince told GEN Edge. "Now, potentially any clinician in the world can chat with Claude and design an antibiotic that may work."

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Max Planck won 1918's Nobel Prize for physics. Yet two of his papers were retracted — a move now being criticized by Yves Gingras, a historian of physics at the University of Quebec and Mahdi Khelfaoui, a fellow historian of science at UQ Trois-Rivières. Science reports: The papers, both quietly retracted in 2011, originally appeared in the early 1940s in Naturwissenschaften, a German journal now owned by publishing giant Springer Nature. After some sleuthing, Khelfaoui determined one of the Planck pieces, a philosophical essay from 1942 titled "Sinn und Grenzen der exakten Wissenschaft" ("Meaning and Limits of Exact Science"), about how to achieve certainty in scientific knowledge, had also appeared in two other journals and been reprinted twice in books. Repackaging the same work multiple times is considered "self-plagiarism" and frowned upon today — the practice produces copyright conflicts and inflates scholars' publication records. The Naturwissenschaften site gives "copyright violation" as the reason for the retraction. Yet publishing identical material in multiple journals was widespread before the internet. "Science was more fragmented" then, Khelfaoui says. "You wanted different audiences ... to have access to your work." The practice was especially common for luminaries like Planck. Albert Einstein did the same (but escaped retractions). Springer Nature's "anachronistic" application of modern standards to a 1942 paper "distort[s] the historical record," Gingras and Khelfaoui argue in a preprint posted last month on arXiv. Any concerns about copyright violations are largely moot anyway: Because Planck died in 1947, his works are in the public domain in most countries. Gingras was especially incensed that Springer Nature deviated from the normal practice of merely slapping the word RETRACTED across the digital version of the paper while still allowing scholars to read the text. Instead, the publisher posted a blank white page with the cryptic phrase, "This article has been withdrawn due to article violation." Springer Nature is nevertheless still selling the empty PDF for $39.95. Suzanne Scarlata, a chemist and biochemist at the Worcester Polytechnic Institute and editor-in-chief of The Science of Nature, as Naturwissenschaften is now known, had not heard about the retractions before being contacted for this story... Scarlata suspects Springer Nature's internal policing software removed the paper and posted the retraction notice unilaterally, without human supervision: "I think it just happened with their algorithm," she says. "It's a mistake they should probably rectify." A second Planck paper was apparently removed because its response to a 1940 paper had used an identical title. Thanks to our long-time Slashdot reader He Who Has No Name for sharing the article.

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alternative_right shares a report from ScienceAlert: If you consumed a wild mushroom and suddenly started seeing tiny people around you, you might reasonably assume it contained a familiar psychedelic. But that does not appear to be the case with Lanmaoa asiatica, known locally as jian shou qing, a mushroom species sold in markets in Yunnan, southwestern China. When eaten undercooked, the mushroom can produce vivid visions of miniature people -- not unlike Gulliver on his travels to Lilliput. To try and find out the root cause, University of Utah mycologists Colin Domnauer and Bryn Dentinger sequenced the genomes of 53 mushroom samples from across the wider Lanmaoa genus. And despite the reported hallucinations, they found no close matches to genes associated with psilocybin or ibotenic acid, two well-known mushroom hallucinogens whose biosynthetic pathways were specifically examined in the study. "Biosynthetic gene mining of the L. asiatica genome found no close hits with any genes known in the production of mushroom psychoactive compounds," write the researchers in their published paper. "This supports our hypothesis of the presence of a novel unidentified metabolite responsible for the unique hallucinogenic properties of L. asiatica." [...] Whatever chemical pathways are causing these effects in the brain, the responsible compound appears to be something scientists have not yet identified. [...] By identifying 1,515 corresponding genes across the selected specimens, the researchers obtained a clearer answer to the question of what defines a mushroom species as part of the genus Lanmaoa. There are now 17 recognized species in the genus, including four that haven't been identified before, two of which the researchers specifically named here: Lanmaoa fallax and Lanmaoa carbonilivor. The researchers say the Lanmaoa family and evolutionary tree can now be more fully mapped out, and some existing specimens may need to be reclassified.

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